Postgraduate research 

Neuroscience & Psychology PhD/iPhD/MD

Travel advice for postgraduate research students

The latest Scottish Government guidance confirms that most students should not plan to travel to term-time accommodation at this point. Where there is a time-sensitive element to your course, a small number of students will be able to travel.

There are some exceptions to this advice, with the following groups of students allowed to be on campus:

  • those who have remained over the winter break
  • those whose attendance is critical and whose education cannot be delivered remotely or postponed, essential placements, or for reasons of student wellbeing

Please note that all arrangements are subject to  future reviews of COVID-19 conditions. We will keep all students updated via email and on this website, as new guidance emerges.

The advice on testing applies to all postgraduate students. If you are travelling to a term-time address you should book a test for the date of your arrival.

Please continue to observe the latest Scottish Government guidance and local restrictions.

  • The advice on testing applies to all PGR students. If you are travelling to a term-time address you should  for the date of your arrival.
  • Please continue to observe the latest Scottish Government guidance and local restrictions.

Travel advice for international students


brain scan and patient

We strive to understand the central nervous system at multiple levels of function, from cells to cognition to social interactions. Our approaches range from molecular, cellular and experimental systems to the brain imaging of human behaviour and cognition as well as social level investigations.

  • PhD: 3-4 years full-time; 5 years part-time;
  • MD (Doctor of Medicine): 2 years full-time; 4 years part-time;

Research projects

Self-funded PhD opportunities

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Functional MRI of visual predictions in cortex

Supervisor: Prof Lars Muckli & Dr Bianca van Kemenade

Project outline: Perception is an active and dynamic process. Our senses are constantly bombarded by sensory input, and in order to make sense of this (sometimes noisy) information, it is thought that we generate predictions that enable us to either enhance the signal to allow for more veridical perception, or to suppress expected input to allow for more resources processing unexpected input. There are still many questions regarding how we generate such sensory predictions, where they are generated in the brain, and how they influence our perception. This project will combine psychophysics and fMRI to answer such questions.

Summary aim: To determine how sensory predictions are generated in the human brain, and how these predictions influence our perception.

Techniques to be used:

  • Brain imaging (fMRI)
  • Behavioural methods (psychophysics)
  • Python programming

References:

  • Petro et al., (2014), Contributions of cortical feedback to sensory processing in primary visual cortex. Frontiers in Psychology
  • Press et al., (2019), The perceptual prediction paradox. Trends in Cognitive Sciences.

Contact:

Prof Lars Muckli, Institute of Neuroscience and Psychology, Lars.Muckli@glasgow.ac.uk, 0141 330 6237 

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Investigation of the somatosensory coding mechanism in the spinal cord

Supervisor: Dr Junichi Hachisuka

Project outline: Somatosensory information including pain and itch is conveyed through the spinal cord to the brain. However, the physiological and anatomical basis of somatosensory processing in the spinal cord is still largely unknown. Our goal is to understand how the spinal dorsal horn neurons differentiate various somatosensory stimuli, especially those associated with pain and itch. To tackle this question, we have developed a semi-intact somatosensory preparation that enables us to record the spinal dorsal horn neuron activity in response to natural stimulation of the skin. Combining this with molecular, genetic and anatomical approaches, we will examine the physiological properties of spinal cord neurons to the natural sensory stimuli.

Summary aim:To reveal the coding mechanism for somatosensory perception including pain and itch in spinal cord dorsal horn neurons.

Techniques to be used:

  • Whole-cell patch clamp recording
  • Retrograde neuronal tracing
  • Immunocytochemistry
  • Neuronal reconstruction
  • Behavioural testing


References:

  • Choi, S. et al. Parallel ascending spinal pathways for affective touch and pain. Nature (2020).
  • Hachisuka, J. et al. Semi-intact ex vivo approach to investigate spinal somatosensory circuits. Elife 5, 1–19 (2016).
  • Hachisuka, J., Koerber, H. R. & Ross, S. E. Selective-cold output through a distinct subset of lamina I spinoparabrachial neurons. Pain 161, 185–194 (2020).
  • Hachisuka, J. et al. Wind-up in lamina I spinoparabrachial neurons: A role for reverberatory circuits. Pain 159, 1484–1493 (2018).
  • Kardon, A. P. et al. Dynorphin acts as a neuromodulator to inhibit itch in the dorsal horn of the spinal cord. Neuron 82, 573–86 (2014).
  • Snyder, L. M. et al. Kappa Opioid Receptor Distribution and Function in Primary Afferents. Neuron 99, 1274-1288.e6 (2018).

Contact:

Dr Junichi Hachisuka (Junichi.Hachisuka@glasgow.ac.uk), Senior Lecturer, Institute of neuroscience and Psychology, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow G12 8QQ

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Synaptic circuitry of the spinal dorsal horn: unmasking the anatomical basis of chronic pain

Dr. David I Hughes

Project outline: Chronic (persistent) pain affects 7.8 million people in the UK, however, only 66% of these patients respond to treatments that are currently available. Our failure to offer effective pain relief for a significant proportion of the population not only presents serious welfare problems, but also serves to highlight how little is known about the anatomical, physiological and pharmacological basis of sensory systems in health and disease. One of the defining characteristics of chronic pain is the development of tactile allodynia, in which previously innocuous mechanical stimuli are perceived as being painful.

This project aims to further our understanding of neuronal circuitry in the spinal dorsal horn by identifying populations of interneurons that receive direct inputs from tactile afferents and have the capacity to activate pain pathways following the development of neuropathic pain.

Summary aim: To identify dorsal horn interneurons implicated in the development of tactile allodynia after peripheral nerve injury. 

Techniques to be used: A variety of molecular, cellular and systems level techniques will be used, including

  • Recovery surgical techniques
  • behavioural testing
  • general histological techniques and immunocytochemistry
  • confocal microscopy
  • transmission electron microscopy
  • image analysis using dedicated software (e.g. Neurolucida for confocal, Neurolucida Explorer, Meta Morph and Image J)

References:

  1. Todd AJ (2010). Neuronal circuitry for pain processing in the dorsal horn. Nature Reviews Neuroscience 11:823-836 (doi:10.1038/nrn2947)
  2. Yasaka T, Tiong SY, Hughes DI, Riddell JS, Todd AJ (2010). Populations of inhibitory and excitatory interneurons in lamina II of the adult rat spinal dorsal horn revealed by a combined electrophysiological and anatomical approach. Pain 151: 475-488 (doi:10.1016/j.pain.2010.08.008)
  3. Hughes DI, Scott DG, Riddell JS, Todd AJ (2007). Upregulation of substance P in low-threshold myelinated afferents is not required for tactile allodynia in the chronic constriction injury and spinal nerve ligation models. Journal of Neuroscience 27: 2025-2034 (doi:10.1523/JNEUROSCI.5401-06.2007)
  4. Hughes DI, Sikander S, Kinnon CM, Boyle KA, Watanabe M, Callister RJ, Graham BA (2012). Morphological, neurochemical and electrophysiological features of parvalbumin-expressing cells: a likely source of axo-axonic inputs in the mouse spinal dorsal horn. Journal of Physiology 590: 3927-3951 (doi:10.1113/jphysiol.2012.235655)
  5. Smith KM, Boyle KA, Madden JF, Dickinson SA, Jobling P, Callister RJ, Hughes DI, Graham BA (2015) Functional heterogeneity of calretinin-expressing neurons in the mouse superficial dorsal horn: implications for spinal pain processing. J Physiol 593: 4319-4339 (doi:10.1113/JP270855)
  6. Abraira VE, Kuehn ED, Chirila AM, Springel MW, Toliver AA, Zimmerman AL, Orefice LL, Boyle KA, Bai L, Song BJ, Bashista KA, O?Neill TG, Zhuo J, Tsan C, Hoynoski J, Rutlin M, Kus L, Niederkofler V, Watanabe M, Dymecki SM, Nelson SB, Heintz N, Hughes DI, Ginty DD (2017) The cellular and synaptic architecture of the mechanosensory dorsal horn Cell 168: 295-310 (doi:10.1016/j.cell.2016.12.010)


 Contact:

  • Dr David I Hughes (David.I.Hughes@glasgow.ac.uk), Senior Lecturer, Institute of Neuroscience and Psychology, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow, G12 8QQ

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Synaptic plasticity deficits in schizophrenia

Supervisor: Prof. Brian Morris

Project outline: Schizophrenia is a common and severe disease with a strong genetic influence. A number of functionally-related genes are believed to interact with environmental factors, such as stress or infection during early development (in utero), to cause the disease. We have recently identified a gene involved in glutamatergic signalling in the CNS that shows strong genetic association with schizophrenia. This gene becomes especially interesting, considering that it is also activated during maternal exposure to infection during pregnancy. This project aims to test the hypothesis that dysfunction in this gene mediates stress and immune influences on disease risk, and contributes to the neurochemical and cognitive impairments of the disease. 

Summary aim: To determine the extent to which the gene is involved in synaptic plasticity in the CNS, and in stress and immune responses, and whether gene deficiency produces cortical GABAergic deficits, impairs glutamatergic signalling or produces behavioural changes characteristic of schizophrenia (deficits in working memory and attentional processing).

Techniques to be used: A variety of molecular, cellular and systems level techniques will be used, including

  • Neuronal and microglial cell cultures
  • gene transfection
  • immunofluorescence
  • RT-PCR
  • western blotting
  • mouse behavioural analysis.

References:

  1. Morris, B. J. and J. A. Pratt (2014). "Novel treatment strategies for schizophrenia from improved understanding of genetic risk." Clinical Genetics. (DOI 10.1111/cge.12485)
  2. Winchester, C. L., H. Ohzeki, et al. (2012). "Converging evidence that sequence variations in the novel candidate gene MAP2K7 (MKK7) are functionally associated with schizophrenia." Human Molecular Genetics 21: 4910-21.
  3. Coffey, E. T. (2014). "Nuclear and cytosolic JNK signalling in neurons." Nature Reviews Neuroscience 15: 285-99
  4. Openshaw, R.L et al., (2019) “JNK signalling mediates aspects of maternal immune activation: importance of maternal genotype in relation to schizophrenia risk” Journal of Neuroinflammation. 16: 18
  5. Bristow, G.C. et al (2020) “16p11 Duplication Disrupts Hippocampal-Orbitofrontal-Amygdala Connectivity, Revealing a Neural Circuit Endophenotype for Schizophrenia” Cell Reports, 31, 107536, https://doi.org/10.1016/j.celrep.2020.107536.

Contact:

  • Prof Brian Morris (Brian.Morris@glasgow.ac.uk),  Professor of Molecular Neurobiology, Institute of Neuroscience and Psychology, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow, G12 8QQ

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Spinal cord neuronal pathways for pain and itch

Supervisor: Prof. Andrew Todd

Project outline: The dorsal horn of the spinal cord plays an important role in processing sensory information that is perceived as pain and itch, but despite its importance we know little about the organisation of neural circuits in this region. Sensory information is conveyed to the brain via projection neurons, which are concentrated in lamina I and scattered through the deeper dorsal horn laminae. However, the vast majority of neurons in laminae I-III are interneurons, with axons that arborise locally. Around two-thirds of these are glutamatergic (excitatory), while the remainder are inhibitory and use GABA and/or glycine.

Recent studies have demonstrated that the inhibitory interneurons can be divided into a number of distinct neurochemical populations that differ in their synaptic inputs and outputs, and this has begun to shed light on how the inhibitory circuits are organised. We offer a variety of projects that will explore the neuronal organisation and synaptic circuitry of the dorsal horn, and how these circuits contribute to the transmission and modulation of sensory information at the spinal level.

Summary aim: To identify distinct functional populations among dorsal horn neurons and to define their roles in synaptic circuits that are responsible for the perception of pain and itch.

Techniques to be used: 

  • immunocytochemistry
  • confocal and electron microscopy
  • neuronal reconstruction
  • retrograde neuronal tracing
  • PCR (for genotyping).

References:

  1. Todd AJ (2010) Neuronal circuitry for pain processing in the dorsal horn. Nature Reviews of Neuroscience 11:823-836.
  2. Polgár E, Sardella TCP, Tiong SYX, Locke S, Watanabe M, Todd AJ (2013) Functional differences between neurochemically-defined populations of inhibitory interneurons in the rat spinal cord. Pain 154:2606-2615.
  3. Kardon AP, Polgár E, Hachisuka J, Snyder LM, Cameron D, Savage S , Cai X, Karnup S, Fan CR,. Hemenway GM, Bernard CS, Schwartz ES, Nagase H, Schwarzer C, Watanabe M, Furuta T, Kaneko T, Koerber HR, Todd AJ, Ross SE (2014) Dynorphin acts as a neuromodulator to inhibit itch in the dorsal horn of the spinal cord. Neuron 82:573-586.

Contact:

  • Prof Andrew J Todd (andrew.todd@glasgow.ac.uk) MBBS, PhD, FSB, Professor of Neuroscience, Institute of Neuroscience and Psychology, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow G12 8QQ;
  • Dr John S Riddell (john.riddell@glasgow.ac.uk) PhD, Institute of Neuroscience and Psychology, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow G12 8QQ

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Neurobiology of reward learning and decision making in humans

Supervisor: Dr. Marios Philiastides

Project outline: Imagine picking wild berries in a forest when suddenly a swarm of bees flies out from behind a bush. In a split second, your motor system has already reacted to flee the swarm. This automatic response constitutes a powerful survival mechanism that allows efficient behaviour switching to escape from a potential hazard in the environment. In turn, a separate and more deliberate process of learning to avoid similar situations will also occur, rendering future berry picking attempts less appealing.

Our lab is interested in understanding the neural pathways and mechanistic principles guiding these processes. To this end we use a multimodal neuroimaging approach in combination with computational modelling and advanced statistical data analysis techniques to closely scrutinise the data collected from human participants.

Our ultimate goal in characterising these neural processes is to further improve our understanding of how everyday responses to rewarding or stressful events can affect our capacity to make optimal decisions, as well as facilitate the study of how mental disorders—such as chronic stress, obsessive-compulsive-disorder, post-traumatic disorder and depression—affect learning and strategic planning. 

Summary aim: Projects are available to investigate the neurobiological basis of reward learning and decision making in humans using multimodal neuroimaging (e.g. MEG/EEG, fMRI, simultaneous EEG/fMRI) and computational modelling. These projects are designed to characterize the patterns of brain activity underlying reward and value-based decisions, the computational principles underlying such decisions, and how these can be used to identify biomarkers for disorders known to compromise ones decision making faculties to be used in future neurobiological or clinical research.

Techniques to be used: 

  • M/EEG
  • fMRI
  • ultra-high field (7T) fMRI
  • simultaneous EEG/fMRI
  • tES
  • pupillometry
  • multivariate pattern analysis
  • computational modelling

References:

  1. Elsa Fouragnan, Chris Retzler, Karen Mullinger and Marios G. Philiastides (2015), Two spatiotemporally distinct value systems shape reward-based learning in the human brain, Nature Communications, 6: 8107.
  2. Marios G. Philiastides, Guido Biele, and Hauke R. Heekeren (2010), A mechanistic account of value computation in the human brain, PNAS, 107 (20): 9430-9435.
  3. Marios G. Philiastides, Guido Biele, Niki Vavatzanidis, Philipp Kazzer and Hauke R. Heekeren (2010), Temporal dynamics of prediction error processing during reward-based decision making, NeuroImage, 53 (1): 221-232.

Contact:

  • Marios Philiastides, Institute of Neuroscience and Psychology, Centre for Cognitive Neuroimaging (CCNi),  University of Glasgow, 58 Hillhead Street, Glasgow G12 8QB, UK. Marios.Philiastides@glasgow.ac.uk Ph: +44 (0) 141 330 4774

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Synaptic plasticity deficits in schizophrenia

Supervisor: Prof. Brian Morris

Project outline: Schizophrenia is a common and severe disease with a strong genetic influence. A number of functionally-related genes are believed to interact with environmental factors, such as stress or infection during early development (in utero), to cause the disease. We have recently identified a gene involved in glutamatergic signalling in the CNS that shows strong genetic association with schizophrenia. This gene becomes especially interesting, considering that it is also activated during maternal exposure to infection during pregnancy. This project aims to test the hypothesis that dysfunction in this gene mediates stress and immune influences on disease risk, and contributes to the neurochemical and cognitive impairments of the disease. 

Summary aim: To determine the extent to which the gene is involved in synaptic plasticity in the CNS, and in stress and immune responses, and whether gene deficiency produces cortical GABAergic deficits, impairs glutamatergic signalling or produces behavioural changes characteristic of schizophrenia (deficits in working memory and attentional processing).

Techniques to be used: A variety of molecular, cellular and systems level techniques will be used, including

  • Neuronal and microglial cell cultures
  • gene transfection
  • immunofluorescence
  • RT-PCR
  • western blotting
  • mouse behavioural analysis.

References:

  1. Morris, B. J. and J. A. Pratt (2014). "Novel treatment strategies for schizophrenia from improved understanding of genetic risk." Clinical Genetics. (DOI 10.1111/cge.12485)
  2. Winchester, C. L., H. Ohzeki, et al. (2012). "Converging evidence that sequence variations in the novel candidate gene MAP2K7 (MKK7) are functionally associated with schizophrenia." Human Molecular Genetics 21: 4910-21.
  3. Coffey, E. T. (2014). "Nuclear and cytosolic JNK signalling in neurons." Nature Reviews Neuroscience 15: 285-99
  4. Openshaw, R.L et al., (2019) “JNK signalling mediates aspects of maternal immune activation: importance of maternal genotype in relation to schizophrenia risk” Journal of Neuroinflammation. 16: 18
  5. Bristow, G.C. et al (2020) “16p11 Duplication Disrupts Hippocampal-Orbitofrontal-Amygdala Connectivity, Revealing a Neural Circuit Endophenotype for Schizophrenia” Cell Reports, 31, 107536, https://doi.org/10.1016/j.celrep.2020.107536.

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Overview

Discovering how the central nervous system functions normally and how it is affected by disease and injury present major challenges for biological and medical research in the 21st century. Over the last two decades there has been an explosion of interest in understanding the normal function of the brain illustrated by launching of the Human Brain Project and the Brain Initiative. In parallel the huge burden of neurological and psychiatric disorders on society and the current lack of effective treatments means there is an urgent need to develop new approaches.

The Institute of Neuroscience & Psychology (INP) has an interactive network that comprises four centres of excellence: Neuroscience, Stroke and Brain Imaging, Cognitive Neuroimaging and Social Interaction.  Via their interactions, our centres aim to understand brain networks at multiple levels of function, from cells to cognition with a strong emphasis on imaging and computational analyses of each level. Our translational efforts are directed at a range of disorders including pain, stroke, spinal cord injury, neurodevelopmental disorder and schizophrenia.

Our staff and students have access to world-class imaging infrastructure and supporting high-performance computing facilities. This provides strong unifying technological and methodological links across the different centres of the INP, including a state-of-the-art platform of cognitive imaging in humans, a high field small bore animal scanner, dedicated confocal and electron microscopy facilities, as well as cutting edge equipment to measure dynamic social signals.

A PhD programme in Neuroscience and Psychology is based on individual research projects covering an exciting range of topics including:

  • non-invasive multimodal brain imaging using fMRI
  • dynamics of auditory and visual processing
  • functions of brain oscillations
  • neuroendocrine effects on social interactions
  • neuromodulation via TMS
  • spinal cord and brainstem circuits in pain
  • molecular mechanisms of synaptic transmission and plasticity
  • neural control of respiration
  • schizophrenia
  • spinal cord injury
  • rett syndrome
  • imaging in acute stroke
  • stroke clinical trial and design
  • stem cells as treatment for stroke
  • experimental stroke
  • cortical circuits mediating perception and memory
  • circadian rhythms

Study options

PhD

  • Duration: 3/4 years full-time; 5 years part-time

Individual research projects are tailored around the expertise of principal investigators.

MD (Doctor of Medicine)

  • Duration: 2 years full-time; 4 years part-time (for medically-qualified graduates only)

Entry requirements

A 2.1 Honours degree or equivalent.

English language requirements

For applicants whose first language is not English, the University sets a minimum English Language proficiency level.

International English Language Testing System (IELTS) Academic module (not General Training)

  • 6.5 with no sub-test under 6.0. 
  • Tests must have been taken within 4 years 5 months of start date. Combined scores from two tests taken within 6 months of each other can be considered.

Common equivalent English language qualifications

All stated English tests are acceptable for admission to this programme:

TOEFL (ib, my best or athome)

  • 90 with minimum R 20, L 19, S 19, W 23. 
  • Tests must have been taken within 4 years 5 months of start date. Combined scores from two tests taken within 6 months of each other can be considered.

PTE (Academic)

  • 60 with minimum 59 in all sub-tests.
  • Tests must have been taken within 4 years 5 months of start date. Combined scores from two tests taken within 6 months of each other can be considered.

Duolingo

  • 120 with 120 in two or more sub-scores including literacy and no subscore below 110 for direct entry, in-sessional support requirement available for those with 120, 100 for 5 week PSE, 100 for 10 week PSE.
  • Tests must have been taken within 1 year of start date.


Glasgow International College English Language (and other foundation providers)

  • 65%.
  • Tests are accepted for academic year following sitting.

University of Glasgow Pre-sessional courses

  • Tests are accepted for academic year following sitting.


Alternatives to English Language qualification

  • Undergraduate degree from English speaking country (including Canada if taught in English)
  • Undergraduate 2+2 degree from English speaking country
  • Undergraduate 2+2 TNE degree taught in English in non-English speaking country
  • Masters degree from English speaking country
  • Masters degree (equivalent on NARIC to UK masters degree) taught in English in non-English speaking country.

For international students, the Home Office has confirmed that the University can choose to use these tests to make its own assessment of English language ability for visa applications to degree level programmes. The University is also able to accept an IELTS test (Academic module) from any of the 1000 IELTS test centres from around the world and we do not require a specific UKVI IELTS test for degree level programmes. We therefore still accept any of the English tests listed for admission to this programme.

Pre-sessional courses

The University of Glasgow accepts evidence of the required language level from the English for Academic Study Unit Pre-sessional courses. We also consider other BALEAP accredited pre-sessional courses:

Fees and funding

Fees

2021/22

  • UK: £4,500
  • International & EU: £23,000

Prices are based on the annual fee for full-time study. Fees for part-time study are half the full-time fee.

Additional fees for all students:

  • Re-submission by a research student £540
  • Submission for a higher degree by published work £1,355
  • Submission of thesis after deadline lapsed £350
  • Submission by staff in receipt of staff scholarship £790

Depending on the nature of the research project, some students will be expected to pay a bench fee (also known as research support costs) to cover additional costs. The exact amount will be provided in the offer letter.

Alumni discount

We offer a 20% discount to our alumni on all Postgraduate Research and full Postgraduate Taught Masters programmes. This includes University of Glasgow graduates and those who have completed Junior Year Abroad, Exchange programme or International Summer School with us. The discount is applied at registration for students who are not in receipt of another discount or scholarship funded by the University. No additional application is required.

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2020/21 fees

  • £4,407 UK/EU
  • £21,920 outside EU

Prices are based on the annual fee for full-time study. Fees for part-time study are half the full-time fee.

Additional fees for all students:

  • Re-submission by a research student £525
  • Submission for a higher degree by published work £1,315
  • Submission of thesis after deadline lapsed £340
  • Submission by staff in receipt of staff scholarship £765

Depending on the nature of the research project, some students will be expected to pay a bench fee (also known as research support costs) to cover additional costs. The exact amount will be provided in the offer letter.

Alumni discount

We offer a 20% discount to our alumni commencing study in Academic session 2020/21, on all Postgraduate Research and full Postgraduate Taught Masters programmes. This includes University of Glasgow graduates and those who have completed a Study Abroad programme or the Erasmus Programme at the University of Glasgow. This discount can be awarded alongside other University scholarships. 

Funding for EU students

The Scottish Government has confirmed that fees for EU students commencing their studies 2020/21 will be at the same level as those for UK student.

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Funding

The iPhD  is not supported by University of Glasgow Scholarship/Funding

Support

The College of Medical, Veterinary and Life Sciences Graduate School provides a vibrant, supportive and stimulating environment for all our postgraduate students. We aim to provide excellent support for our postgraduates through dedicated postgraduate convenors, highly trained supervisors and pastoral support for each student.
 
Our overarching aim is to provide a research training environment that includes:

  • provision of excellent facilities and cutting edge techniques
  • training in essential research and generic skills
  • excellence in supervision and mentoring
  • interactive discussion groups and seminars
  • an atmosphere that fosters critical cultural policy and research analysis
  • synergy between research groups and areas
  • extensive multidisciplinary and collaborative research
  • extensive external collaborations both within and beyond the UK 
  • a robust generic skills programme including opportunities in social and commercial training

Many of our project supervisors have strong academic connections with international collaborators in universities and research institutes across the world. Funds are available through the college of Medical, Veterinary and Life Sciences to allow visits to international laboratories where part of your project can be carried out, if you and your supervisor decide this would enhance your research and training. This provides an excellent opportunity for networking and increasing your scientific knowledge and skill set. Some supervisors also have strong links with industry. The university organises an open day to highlight career opportunities in industry related to our research.

Resources

We offer a wide range of cutting-edge research facilities. Our imaging centre is equipped with state of the art technology for multimodal human brain imaging (fMRI, MEG, TMS and EEG) with sophisticated analysis methods to study the functioning of the human brain. In the near future we will extend these facilities to include the unique Imaging Centre of Excellence at the new South Glasgow University Hospital which will include a world-leading £7m ultra high-field MRI scanner, a facility which will be unique in the UK.

Our laboratories have a wide range of resources and technical expertise for studies in experimental systems using cell culture, confocal microscopy, gene therapy, electrophysiology, 7 Tesla small bore experimental MRI, behavioural assessment. 

How to apply

Identify potential supervisors

All Postgraduate Research Students are allocated a supervisor who will act as the main source of academic support and research mentoring. You may want to identify a potential supervisor and contact them to discuss your research proposal before you apply. Please note, even if you have spoken to an academic staff member about your proposal you still need to submit an online application form.

You can find relevant academic staff members with our staff research interests search.


Gather your documents

Before applying please make sure you gather the following supporting documentation:

  1. Final or current degree transcripts including grades (and an official translation, if needed) – scanned copy in colour of the original document.
  2. Degree certificates (and an official translation, if needed): scanned copy in colour of the original document.
  3. Two references on headed paper and signed by the referee. One must be academic, the other can be academic or professional. References may be uploaded as part of the application form or you may enter your referees contact details on the application form. We will then email your referee and notify you when we receive the reference.  We can also accept confidential references direct to rio-researchadmissions@glasgow.ac.uk, from the referee’s university or business email account.
  4. Research proposal, CV, samples of written work as per requirements for each subject area.

Apply now

I've applied. What next?

If you have any other trouble accessing Applicant Self-Service, please see Application Troubleshooting/FAQs. 


Contact us

Before you apply

PhD/MSc/MD: email mvls-gradschool@glasgow.ac.uk

iPhD: email mvls-iphd@glasgow.ac.uk

After you have submitted your application

PhD/MSc/MD/iPhD: contact our Admissions team

Any references may be submitted by email to: rio-researchadmissions@glasgow.ac.uk