Dr Denggao Yao

  • Postdoctoral Research Associate (Immunology)

telephone: 01413308389
email: Denggao.Yao@glasgow.ac.uk

B334, Iii - Gbrc,, Sir Graeme Davies Building, 120 University Place, Glasgow, G12 8ta

Publications

List by: Type | Date

Jump to: 2021 | 2020 | 2019 | 2018 | 2016 | 2015 | 2014 | 2013 | 2009 | 2008 | 2006 | 2005
Number of items: 19.

2021

McGonigal, R., Barrie, J. A., Yao, D., Black, L. E. , McLaughlin, M. and Willison, H. J. (2021) Neuronally expressed a-series gangliosides are sufficient to prevent the lethal age-dependent phenotype in GM3-only expressing mice. Journal of Neurochemistry, 158(2), pp. 217-232. (doi: 10.1111/jnc.15365) (PMID:33864399)

2020

Cunningham, M. E. , Meehan, G. R. , Robinson, S., Yao, D., McGonigal, R. and Willison, H. J. (2020) Perisynaptic Schwann cells phagocytose nerve terminal debris in a mouse model of Guillain-Barré syndrome. Journal of the Peripheral Nervous System, 25(2), pp. 143-151. (doi: 10.1111/jns.12373) (PMID:32250537)

2019

McGonigal, R., Barrie, J.A., Yao, D., McLaughlin, M. , Cunningham, M.E. , Rowan, E.G. and Willison, H.J. (2019) Glial sulfatides and neuronal complex gangliosides are functionally interdependent in maintaining myelinating axon integrity. Journal of Neuroscience, 39(1), pp. 63-77. (doi: 10.1523/JNEUROSCI.2095-18.2018) (PMID:30446529) (PMCID:PMC6325269)

2018

Meehan, G. R. , McGonigal, R., Cunningham, M. E. , Wang, Y., Barrie, J. A., Halstead, S. K., Gourlay, D., Yao, D. and Willison, H. J. (2018) Differential binding patterns of anti-sulfatide antibodies to glial membranes. Journal of Neuroimmunology, 323, pp. 28-35. (doi: 10.1016/j.jneuroim.2018.07.004) (PMID:30196830) (PMCID:PMC6134133)

2016

Cunningham, M. , McGonigal, R., Meehan, G. R. , Barrie, J. A., Yao, D., Halstead, S. K. and Willison, H. J. (2016) Anti-ganglioside antibodies are removed from circulation in mice by neuronal endocytosis. Brain, 139(6), pp. 1657-1665. (doi: 10.1093/brain/aww056) (PMID:27017187) (PMCID:PMC4892750)

McGonigal, R., Cunningham, M. E. , Yao, D., Barrie, J. A., Sankaranarayanan, S., Fewou, S. N., Furukawa, K., Yednock, T. A. and Willison, H. J. (2016) C1q-targeted inhibition of the classical complement pathway prevents injury in a novel mouse model of acute motor axonal neuropathy. Acta Neuropathologica Communications, 4, 23. (doi: 10.1186/s40478-016-0291-x) (PMID:26936605) (PMCID:PMC4776408)

2015

Delmont, E., Robb, H., Davidson, A., Halstead, S., Yao, D., Meehan, G. R. and Willison, H. (2015) Prospective study comparing enzyme-linked immunosorbent assay and glycoarray assay to detect antiglycolipid antibodies in a routine diagnostic neuroimmunology laboratory setting. Clinical and Experimental Neuroimmunology, 6(2), pp. 175-182. (doi: 10.1111/cen3.12193)

Delmont, E., Halstead, S., Galban-Horcajo, F., Yao, D., Desnuelle, C. and Willison, H. (2015) Improving the detection of IgM antibodies against glycolipids complexes of GM1 and galactocerebroside in multifocal motor neuropathy using glycoarray and ELISA assays. Journal of Neuroimmunology, 278, pp. 159-161. (doi: 10.1016/j.jneuroim.2014.11.001) (PMID:25468269)

2014

Macdonald, F., Yao, D., Quinn, J. and Greenhalgh, D. (2014) PTEN ablation in RasHa/Fos skin carcinogenesis invokes p53-dependent p21 to delay conversion while p53-independent p21 limits progression via cyclin D1/E2 inhibition. Oncogene, 33(32), pp. 4132-4143. (doi: 10.1038/onc.2013.372)

Yao, D. et al. (2014) Neuronal expression of galnac transferase is sufficient to prevent the age-related neurodegenerative phenotype of complex ganglioside-deficient mice. Journal of Neuroscience, 34(3), pp. 880-891. (doi: 10.1523/JNEUROSCI.3996-13.2014)

2013

Rupp, A., Cunningham, M. E. , Yao, D., Furukawa, K. and Willison, H. J. (2013) The effects of age and ganglioside composition on the rate of motor nerve terminal regeneration following antibody-mediated injury in mice. Synapse, 67(7), pp. 382-389. (doi: 10.1002/syn.21648) (PMID:23401234) (PMCID:PMC4495252)

Rupp, A., Yao, D., Meehan, G. R. and Willison, H. J. (2013) Thy-1 Mice Lend Themselves for the Modelling of Anti-ganglioside Antibody-mediated Disorders. PNS Biennial Meeting of the Peripheral Nerve Society, St Malo, France, 11-14 Jun 2013.

Macdonald, F.H., Yao, D., Quinn, J.A. and Greenhalgh, D.A. (2013) Activated p-AKT, but not MDM2, drives malignant progression in Ras/Fos/PTENnull skin carcinogenesis via p53/p21 loss and elevated cyclin D1/E2 expression. Journal of Investigative Dermatology, 133(S1), S57-S57. (doi: 10.1038/jid.2013.96)

2009

Greenhalgh, D. , Yao, D., Quinn, J. and Macdonald, F. (2009) RasHa activation interdicts compensatory p53/p21WAF expression in Fos/PTENnull skin carcinogenesis via initial p53 loss but retention of p21WAF limits malignant progression. In: American Association for Cancer Research (AACR) 100th Annual Meeting, Denver, USA, 18-22 April 2009,

Macdonald, F., Yao, D., Quinn, J. and Greenhalgh, D. (2009) Compensatory p53/p21WAF-induced differentiation in Fos/PTENnull skin carcinogenesis is interdicted by RasHa activation but retention of p21WAF inhibits malignant progression. In: British Society for Investigative Dermatology (BSID) Annual Meeting, Royal Agricultural College, Cirencester, UK, 30 March - 1 April 2009,

2008

Yao, D., Alexander, C., Quinn, J. , Chan, W., Wu, H. and Greenhalgh, D. (2008) Fos cooperation with PTEN loss elicits keratoacanthoma not carcinoma, owing to p53/p21(WAF)-induced differentiation triggered by GSK3 beta inactivation and reduced AKT activity. Journal of Cell Science, 121(10), pp. 1758-1769.

2006

Yao, D., Alexander, C.L., Quinn, J.A., Porter, M.J., Wu, H. and Greenhalgh, D.A. (2006) PTEN loss promotes rasHa-mediated papillomatogenesis via dual up-regulation of AKT activity and cell cycle deregulation but malignant conversion proceeds via rasHa-independent pathways. Cancer Research, 66(3), pp. 1302-1312.

Shaw, M., Yao, D., Quinn, J.A. and Greenhalgh, D.A. (2006) Paradoxical maintenance of E-cadherin following inducible PTEN ablation in Ras or Fos-mediated skin carcinogenesis identifies sentinel mechanisms geared to combat PTEN dysfunction. In: British Society for Investigative Dermatology Annual Meeting, Manchester, 10-12 April 2006, p. 247. (doi:10.1111/j.1365-2133.2006.07327.x)

2005

Yao, D., Quinn, J.A. and Greenhalgh, D.A. (2005) Inducible cre-mediated N-ras activation and PTEN inactivation in transgenic mouse melanocytes requires keratinocyte hyperplasia to elicit a melanocyte pathology. In: Montagne Symposia, 2004 and 6th International Skin Cancer Conference, 2004, Salishan Resort, Newport, Oregon, USA, Oct 2004, p. 171.

This list was generated on Sat Oct 23 18:06:00 2021 BST.
Number of items: 19.

Articles

McGonigal, R., Barrie, J. A., Yao, D., Black, L. E. , McLaughlin, M. and Willison, H. J. (2021) Neuronally expressed a-series gangliosides are sufficient to prevent the lethal age-dependent phenotype in GM3-only expressing mice. Journal of Neurochemistry, 158(2), pp. 217-232. (doi: 10.1111/jnc.15365) (PMID:33864399)

Cunningham, M. E. , Meehan, G. R. , Robinson, S., Yao, D., McGonigal, R. and Willison, H. J. (2020) Perisynaptic Schwann cells phagocytose nerve terminal debris in a mouse model of Guillain-Barré syndrome. Journal of the Peripheral Nervous System, 25(2), pp. 143-151. (doi: 10.1111/jns.12373) (PMID:32250537)

McGonigal, R., Barrie, J.A., Yao, D., McLaughlin, M. , Cunningham, M.E. , Rowan, E.G. and Willison, H.J. (2019) Glial sulfatides and neuronal complex gangliosides are functionally interdependent in maintaining myelinating axon integrity. Journal of Neuroscience, 39(1), pp. 63-77. (doi: 10.1523/JNEUROSCI.2095-18.2018) (PMID:30446529) (PMCID:PMC6325269)

Meehan, G. R. , McGonigal, R., Cunningham, M. E. , Wang, Y., Barrie, J. A., Halstead, S. K., Gourlay, D., Yao, D. and Willison, H. J. (2018) Differential binding patterns of anti-sulfatide antibodies to glial membranes. Journal of Neuroimmunology, 323, pp. 28-35. (doi: 10.1016/j.jneuroim.2018.07.004) (PMID:30196830) (PMCID:PMC6134133)

Cunningham, M. , McGonigal, R., Meehan, G. R. , Barrie, J. A., Yao, D., Halstead, S. K. and Willison, H. J. (2016) Anti-ganglioside antibodies are removed from circulation in mice by neuronal endocytosis. Brain, 139(6), pp. 1657-1665. (doi: 10.1093/brain/aww056) (PMID:27017187) (PMCID:PMC4892750)

McGonigal, R., Cunningham, M. E. , Yao, D., Barrie, J. A., Sankaranarayanan, S., Fewou, S. N., Furukawa, K., Yednock, T. A. and Willison, H. J. (2016) C1q-targeted inhibition of the classical complement pathway prevents injury in a novel mouse model of acute motor axonal neuropathy. Acta Neuropathologica Communications, 4, 23. (doi: 10.1186/s40478-016-0291-x) (PMID:26936605) (PMCID:PMC4776408)

Delmont, E., Robb, H., Davidson, A., Halstead, S., Yao, D., Meehan, G. R. and Willison, H. (2015) Prospective study comparing enzyme-linked immunosorbent assay and glycoarray assay to detect antiglycolipid antibodies in a routine diagnostic neuroimmunology laboratory setting. Clinical and Experimental Neuroimmunology, 6(2), pp. 175-182. (doi: 10.1111/cen3.12193)

Delmont, E., Halstead, S., Galban-Horcajo, F., Yao, D., Desnuelle, C. and Willison, H. (2015) Improving the detection of IgM antibodies against glycolipids complexes of GM1 and galactocerebroside in multifocal motor neuropathy using glycoarray and ELISA assays. Journal of Neuroimmunology, 278, pp. 159-161. (doi: 10.1016/j.jneuroim.2014.11.001) (PMID:25468269)

Macdonald, F., Yao, D., Quinn, J. and Greenhalgh, D. (2014) PTEN ablation in RasHa/Fos skin carcinogenesis invokes p53-dependent p21 to delay conversion while p53-independent p21 limits progression via cyclin D1/E2 inhibition. Oncogene, 33(32), pp. 4132-4143. (doi: 10.1038/onc.2013.372)

Yao, D. et al. (2014) Neuronal expression of galnac transferase is sufficient to prevent the age-related neurodegenerative phenotype of complex ganglioside-deficient mice. Journal of Neuroscience, 34(3), pp. 880-891. (doi: 10.1523/JNEUROSCI.3996-13.2014)

Rupp, A., Cunningham, M. E. , Yao, D., Furukawa, K. and Willison, H. J. (2013) The effects of age and ganglioside composition on the rate of motor nerve terminal regeneration following antibody-mediated injury in mice. Synapse, 67(7), pp. 382-389. (doi: 10.1002/syn.21648) (PMID:23401234) (PMCID:PMC4495252)

Macdonald, F.H., Yao, D., Quinn, J.A. and Greenhalgh, D.A. (2013) Activated p-AKT, but not MDM2, drives malignant progression in Ras/Fos/PTENnull skin carcinogenesis via p53/p21 loss and elevated cyclin D1/E2 expression. Journal of Investigative Dermatology, 133(S1), S57-S57. (doi: 10.1038/jid.2013.96)

Yao, D., Alexander, C., Quinn, J. , Chan, W., Wu, H. and Greenhalgh, D. (2008) Fos cooperation with PTEN loss elicits keratoacanthoma not carcinoma, owing to p53/p21(WAF)-induced differentiation triggered by GSK3 beta inactivation and reduced AKT activity. Journal of Cell Science, 121(10), pp. 1758-1769.

Yao, D., Alexander, C.L., Quinn, J.A., Porter, M.J., Wu, H. and Greenhalgh, D.A. (2006) PTEN loss promotes rasHa-mediated papillomatogenesis via dual up-regulation of AKT activity and cell cycle deregulation but malignant conversion proceeds via rasHa-independent pathways. Cancer Research, 66(3), pp. 1302-1312.

Conference or Workshop Item

Rupp, A., Yao, D., Meehan, G. R. and Willison, H. J. (2013) Thy-1 Mice Lend Themselves for the Modelling of Anti-ganglioside Antibody-mediated Disorders. PNS Biennial Meeting of the Peripheral Nerve Society, St Malo, France, 11-14 Jun 2013.

Conference Proceedings

Greenhalgh, D. , Yao, D., Quinn, J. and Macdonald, F. (2009) RasHa activation interdicts compensatory p53/p21WAF expression in Fos/PTENnull skin carcinogenesis via initial p53 loss but retention of p21WAF limits malignant progression. In: American Association for Cancer Research (AACR) 100th Annual Meeting, Denver, USA, 18-22 April 2009,

Macdonald, F., Yao, D., Quinn, J. and Greenhalgh, D. (2009) Compensatory p53/p21WAF-induced differentiation in Fos/PTENnull skin carcinogenesis is interdicted by RasHa activation but retention of p21WAF inhibits malignant progression. In: British Society for Investigative Dermatology (BSID) Annual Meeting, Royal Agricultural College, Cirencester, UK, 30 March - 1 April 2009,

Shaw, M., Yao, D., Quinn, J.A. and Greenhalgh, D.A. (2006) Paradoxical maintenance of E-cadherin following inducible PTEN ablation in Ras or Fos-mediated skin carcinogenesis identifies sentinel mechanisms geared to combat PTEN dysfunction. In: British Society for Investigative Dermatology Annual Meeting, Manchester, 10-12 April 2006, p. 247. (doi:10.1111/j.1365-2133.2006.07327.x)

Yao, D., Quinn, J.A. and Greenhalgh, D.A. (2005) Inducible cre-mediated N-ras activation and PTEN inactivation in transgenic mouse melanocytes requires keratinocyte hyperplasia to elicit a melanocyte pathology. In: Montagne Symposia, 2004 and 6th International Skin Cancer Conference, 2004, Salishan Resort, Newport, Oregon, USA, Oct 2004, p. 171.

This list was generated on Sat Oct 23 18:06:00 2021 BST.